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McCall, Charles
One or more keywords matched the following items that are connected to McCall, Charles
Item TypeName
Academic Article Lipopolysaccharide- and lipoteichoic acid-induced tolerance and cross-tolerance: distinct alterations in IL-1 receptor-associated kinase.
Academic Article Distinct post-receptor alterations generate gene- and signal-selective adaptation and cross-adaptation of TLR4 and TLR2 in human leukocytes.
Academic Article Induction of RelB participates in endotoxin tolerance.
Academic Article Chromatin-specific remodeling by HMGB1 and linker histone H1 silences proinflammatory genes during endotoxin tolerance.
Academic Article The NF-kappaB factor RelB and histone H3 lysine methyltransferase G9a directly interact to generate epigenetic silencing in endotoxin tolerance.
Academic Article Facultative heterochromatin formation at the IL-1 beta promoter in LPS tolerance and sepsis.
Academic Article MicroRNA-146a regulates both transcription silencing and translation disruption of TNF-a during TLR4-induced gene reprogramming.
Concept Immune Tolerance
Academic Article MicroRNA-146a and RBM4 form a negative feed-forward loop that disrupts cytokine mRNA translation following TLR4 responses in human THP-1 monocytes.
Academic Article SIRT1 inhibition during the hypoinflammatory phenotype of sepsis enhances immunity and improves outcome.
Academic Article Processing Body Formation Limits Proinflammatory Cytokine Synthesis in Endotoxin-Tolerant Monocytes and Murine Septic Macrophages.
Academic Article Frontline Science: Myeloid cell-specific deletion of Cebpb decreases sepsis-induced immunosuppression in mice.
Academic Article Expression of C/EBPß in myeloid progenitors during sepsis promotes immunosuppression.
Academic Article Sirtuin1 Targeting Reverses Innate and Adaptive Immune Tolerance in Septic Mice.
Academic Article Mitochondrial Sirtuin 4 Resolves Immune Tolerance in Monocytes by Rebalancing Glycolysis and Glucose Oxidation Homeostasis.
Academic Article Nfia deletion in myeloid cells blocks expansion of myeloid-derived suppressor cells during sepsis.
Academic Article Epigenetic and metabolic programming of innate immunity in sepsis.
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  • Immune Tolerance